VANDANA REDDY

BIMONTHLY INTERNAL ASSESSMENT FOR THE MONTH OF JANUARY 2021







January bimonthly assessment exam
2021



       26yr old women came with
complaints of altered sensorium since 1 day,headache since 8 days,fever and vomitings since 4 days

More here: https://harikachindam7.blogspot.com/2020/12/26-year-old-female-with-complaints-of.html

Case presentation  links: 

https://youtu.be/fz9Jssoc-mA

https://youtu.be/d4lLX04oL8s

https://youtu.be/CSCxw2zp7Oc

Pt has complaints since 1 month she has headache 1-2 times per week relieved on medication .but the headache worsened in the past 15 days ., 

c/o Heaeache since 8 days- in bitemporal vertex region 

has been aggrevated since 1 week .

After stopping using steroids she has

fever since 4 days- low grade not associated with chills and rigors.

 vomitings since 1 week (multiple episodes).

She has generalised weakness,decreased apetite and unable to walk for 

Neck pain since 4 days

At the time of presentation she has altered sensorium with irrelevant speech since 3 am in the morning

The anatomical location for her problems could be in meninges or brain parenchyma




1.Hypnatremia secondary to SIADH
2.tubercular meningitis 
3.infarct left thalami region 
4.multiple progressive joint pains due to SLE
b) What is the etiology of the current problem and how would you as a member of the treating team arrive at a diagnosis? Please chart out the sequence of events timeline between the manifestations of each of her problems and current outcomes. 

1) Hyponatremia.

2) Euvolemic Hyponatremia ( as there was no signs of fluid overload or hypotension) secondary to SIADH.(SIADH secondary to TUBERCULAR MENINGITIS).

3) TUBERCULAR MENINGITIS. ( CSF cbnaat came positive and also cell count revealed lymphocytosis,with  low glucose and chloride and increased CSF protein. - evident of TB meningitis.

4) ACUTE INFARCT IN LEFT THALAMUS (secondary to  vasculitis).

Sequence of events:

2010: marriage
!
2011: 1st child birth (male)
!
2012: 2nd child birth (female)
!
2014: Both hands small joints pain progressed to elbow and shoulder - relieved with treatment @ local RMP
!
2017: Multiple joint pains - diagnosed as SLE & started on treatment
!
2020 (december): - headache on& off 1-2 times/week relieved with treatment
!
Increased headache from 15 days
!
10 days back stopped methyl prednisolone
!
 vomitings from 1 week
!
Fever, neckpain, generalized weakness & decreased appetite from 4 days
!
Admitted to a local hospital and @ night went to bathroom and fell down in a state of altered sensorium
!
Same day presented to our casualty @ around
3 am in a state of altered sensorium & irrelevant speech

c) What is the efficacy of each of the drugs listed in her prior treatment plan that she was following since last two years before she stopped it two weeks back? 

Patient was on medication for SLE (Hydrochloroquine-200mg/OD,Sulfasalazine,Methylprednisolone,Alandronic acid and Cholecalciferol,Aceclofenac,Flupirtine,Gabapentine,Methylcobalamin tablets), which she stopped 10 days back.

https://pubmed.ncbi.nlm.nih.gov/8815753/


HCQ therapy is effective on clinical improvement of SLE patients through interfering with inflammatory signaling pathways, reducing anti-DNA autoantibodies and normalizing the complement activity.

https://pubmed.ncbi.nlm.nih.gov/29987550/#:~:text=Conclusion%3A%20HCQ%20therapy%20is%20effective,and%20normalizing%20the%20complement%20activity

d) Why was she given bisphosphonates? 
What is the efficacy of using primary bisphosphonate prophylaxis for patients started on corticosteroids?

To assess the benefits and harms of bisphosphonates for the prevention and treatment of Glucocorticoid induced osteoporosis in adults.



https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6461188/?report=reader


It included 

1. Prevention or treatment of Glucocorticoid induced osteoporosis

2. Adults taking a mean steroid dose of 5.0 mg/day or active treatment including bisphosphonates of any type alone or in combination with calcium or vitamin D

 4) comparator treatment including a control of calcium or vitamin D, or both, alone or with placebo

Patients - 

A total of 27 RCTs with 3075 participants in the review 


- Pooled analysis for incident vertebral fractures included 12 trials (1343 participants)

 Outcome - In this analysis 46/597 people experienced new vertebral fractures in the control group compared with 31/746 in the bisphosphonate group; relative improvement of 43% with bisphosphantes.



Basically BIPHOSPHONATES help in reducing osteoporosis

 when used with steroids


e) What is the efficacy of using primary PPI prophylaxis during initiation of any corticosteroids to prevent Gi ulcers? 

It is a common practice to co-prescribe PPI alongside glucocorticoid therapy, to taper the theoretical risk of peptic ulcer associated with glucocorticoid therapy. However, peptic ulcer is a rare complication of systemic glucocorticoid therapy and occurs in less than 0.4–1.8% of patients. With such low risks, there is no indication for routine prophylaxis with PPIs in this situation.

Any reports of normal  csf leukocyte count and normal csf protein in meningitis? 




What could be the probable cause for a normal csf leukocyte count in a patient with chronic meningitis? 

Meningitis in the absence of pleocytosis on CSF is rare. If this occurs, causative organism is likely bacterial. 

Please share any reports around similar patients with SLE and TB meningitis?


Here's a case report of a 31 year old woman diagnosed with SLE and subsequently developed SLE myocarditis and Lupus Nephritis who also developed TB meningitis


https://pubmed.ncbi.nlm.nih.gov/28764615/


Another case report of a 51 year old woman with SLE who later developed TB meningitis

https://pubmed.ncbi.nlm.nih.gov/10067053/


e) What is the sensitivity and specificity of ANA in the diagnosis of SLE? 


To determine the sensitivity and specificity of ANA and anti-dsDNA in SLE patients, using sera from Healthy controls and Multiple Medical problem patients.

The prevalence of ANA at a titer of ≥1:80 and ≥ 1:160 was 8% and 4%, respectively, in HC; and it was 12% and 6% respectively, in MMP patients. The prevalence of anti-dsDNA was 0% in HC and 3% in MMP patients. When using HC sera for the diagnosis of SLE, the sensitivity of ANA at a titer of ≥ 1:80 and ≥ 1:160 was 98% and 90%, respectively, with specificity of 92% and 96%, respectively. The specificity decreased to 88% and 94%, respectively, when using sera from MMP patients. The specificity of anti-dsDNA was 100% and 97%, when using sera from HC and MMP patients, respectively.

ANA and anti-dsDNA gave high sensitivity and high specificity in patients with SLE, even when using MMP patient's sera as controls.


https://pubmed.ncbi.nlm.nih.gov/24383972/


2.What was the research question in the above thesis presentation? 


Could hypomagnesemia in type 2 DM lead to further more complications


b) What was the researcher's hypothesis? 


According to the presenter, hypomagnesemia in type 2 diabetes mellitus could lead to further more complications and poor prognosis.



c)What is the current available evidence for magnesium deficiency leading to poorer outcomes in patients with diabetes? 


Association of serum magnesium with Type 2 Diabetes Mellitus






Oral Magnesium Supplementation Improves Insulin Sensitivity and Metabolic Control in Type 2 Diabetic Subjects

https://care.diabetesjournals.org/content/26/4/1147#:~:text=In%20conclusion%2C%20oral%20magnesium%20supplementation,thus%20contributing%20to%20metabolic%20control.




https://youtu.be/jXVS5J1-RNE


3 .a)What was the research question in the above thesis presentation? 


b)What was the researcher's hypothesis?


24 hours urinary sodium in newly diagnosed hypertensive patients 

The hypothesis is that, the amount of salt consumed is almost equal to the sodium excreted in the urine, which could lead to increase in the blood pressure



c)What is the current available evidence for the utility of monitoring salt excretion in the hypertensive population? 


Estimation of 24 hours urinary sodium excretion and relation to cardiovascular events






Salt restriction would certainly reduce the blood pressure and 24 hours urinary excretion of sodium does sound like a good way in measuring the salt intake but also it is important for blood pressure monitoring over 24 hours.




3.What is the efficacy of aspirin in stroke in your assessment of the evidence provided in the article. Please go through the RCT CASP checklist here https://casp-uk.net/casp-tools-checklists/ and answer the questions mentioned in the checklist in relation to your article.


Aspirin in the prevention of progressing stroke: a randomized controlled study

 

https://onlinelibrary.wiley.com/doi/full/10.1111/j.1365-2796.2003.01233.x



O - The main results of the trial showed that aspirin treatment did not significantly reduce the rate of stroke progression. The progression rate was 15.9% amongst patients treated with aspirin and 16.7% for those on placebo. In the aspirin group, the relative risk was 0.95 (95% CI 0.62–1.45).


4.Please mention your individual learning experiences from this month.
1.i have seen a lady with frontal lobe mass with altered consciousness 
2.Chronic pancreatitis? Alcohol dependence syndrome 
3. An elderly male Chronic obstructive pulmonary disease
4.A lady with chronic kidney disease on mhd with ulcer over left foot
5. i learned how to do plural tapping
interpretation of ecg 
interpretation of LFT report

6.ihave seen cases like

         .SLE with Tubercular meningitis

         .Pancytopenia 

       .liver abscess with plural effusion 

       .peripheral vascular disease with                     supraventricular tachycardia
         .pericardial effusion


5) a) What are the possible reasons for the 36 year old man's hypertension and CAD described in the link below since three years? 


https://vamsikrishna1996.blogspot.com/2021/01/36-year-male-presented-to-casualty-at.html?m=1


The 36 year old man with CAD, the etiological reasons to his CAD could be due to the long standing hypertension since 3 years along with being an alcoholic and smoker since 15 years adds up to the risk factors.


b) Please describe the ECG changes and correlate them with the patient's current diagnosis. 


The initial ecgs shows irregular rhythm along with VPS

There are progressive ST segment changes in the anteroseptal leads - ? STEMI involving LAD territory/ LAD aneurysm


c) Share an RCT that provides evidence for the efficacy of primary PTCA in acute myocardial infarction over medical management. Describe the efficacy in a PICO format. 

jamanetwork.com/journals/jama/fullarticle/194837


Thrombolytic Therapy vs Primary Percutaneous Coronary Intervention for Myocardial Infarction in Patients Presenting to Hospitals Without On-site Cardiac SurgeryA Randomized Controlled TrialP -  Four hundred fifty-one thrombolytic-eligible patients with acute MI of less than 12 hours' duration associated with ST-segment elevation on electrocardiogram.

 225 received primary PTC

226 participants received accelerated tissue plasminogen activator (bolus dose of 15 mg and an infusion of 0.75 mg/kg for 30 minutes followed by 0.5 mg/kg for 60 minute

 The incidence of the composite end point was reduced in the primary PCI group at 6 weeks (10.7% vs 17.7%) and 6 months (12.4% vs 19.9%) after index MI. Six-month rates for individual outcomes were 6.2% vs 7.1% for death, 5.3% vs 10.6% for recurrent MI, and 2.2% vs 4.0% for stroke for primary PCI vs thrombolytic therapy, respectively. Median length of stay was also reduced in the primary PCI group (4.5 vs 6.0 days)

Conclusions Compared with thrombolytic therapy, treatment of patients with primary PCI  with better clinical outcomes for 6 months after index MI and a shorter hospital stay..sA


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